Captopril
Examined the effects of treatment with an anti-inflammatory p38 MAP kinase inhibitor SB-281832 ; in the N-Nitro-L-arginine methyl ester L-NAME ; -spontaneously hypertensive rat SHR ; model of severe hypertension and accelerated target organ damage. SHRs were dived into control n 10 ; , L-NAME n 16 ; and L-NAME + SB-281832 n 16 ; groups. L-NAME was delivered by the drinking water ad lib 50 mg L ; and SB 281832 was administered by the diet 1500 ppm ; for the duration of L-NAME treatment 3 weeks ; . All baseline renal and hemodynamic measures were similar in all groups. Treatment with SB-281832 significantly reduced micro-albuminuria 32248 vs. 16537 mg dL, p 0.05 ; and chamber constriction ESV of 0.120.10 vs. 0.230.11; p 0.05 and EDV of 0.240.05 vs. 0.410.05; p 0.05 ; . LV thickening was increased with L-NAME treatment LV mass b.w. of 2.50.10 vs. 3.240.15; p 0.05 and relative wall thickness of 0.570.03 vs. 1.370.36 ; . However, mean BP 13217 vs. 17323 bpm ; , as well as, endothelialdependent EC50 701266 vs. 523159 nM carbachol ; and independent EC50 355 vs. 303 nM phenylephrine ; vascular reactivity were not altered by SB-281832 treatment L-NAME vs. treated, respectively ; . Conclusion: The accelerated target organ damage induced by nitric oxide synthase inhibition in the SHR was attenuated by treatment with a p38 MAP kinase inhibitor, SB 281832. The organ protection observed in the heart and kidney was not dependent upon changes in blood pressure or preservation of endothelial function. CAPTOPRIL BLUNTS CARDIAC REMODELING, PREVENTS CARDIAC DYSFUNCTION, AND IMPROVES SURVIVAL IN TWO DIFFERENT MURINE MODELS OF HEART FAILURE. Weike Bao * , Thomas R Schaeffer, Stephen C Lenhard, and Tom C-C Hu. Investigative & Cardiac Biology, CVU CEDD, GlaxoSmithKline, King of Prussia, PA. Clinical trials and animal studies have demonstrated the dramatic benefits from angiotensin-converting enzyme ACE ; inhibition in preventing the cardiovascular diseases. However, the effect of captopril in murine model of transverse aortic banding TAB ; and myocardial infarction MI ; has not been well characterized so far, although both models have been widely used to study the interplay of genes and pathophysiology of cardiac hypertrophy and heart failure in the gene engineered mice. In present study, we assess the long-term effects of attenuating cardiac remodeling and improving cardiac function by using captopril in two different murine model of heart failure. CD1 mice were subjected to TAB and MI, and divided into five groups. Sham n 12 ; , MI TAB n 20 ; , MI captopril n 10 ; , TAB + captopril n 12 ; , captopril 1 g liter of drinking water ; . Cardiac remodeling and function were evaluated under isoflurane anaesthesia by cine-magnetic resonance imaging. MI significantly reduced EF by 66.4% p 0.01 vs. sham ; . In contrast, treatment with captopril increased EF by 29.3% p 0.05 ; compared with untreated mice. TAB resulted in a significant increase in LV mass by 37.2 % p 0.01 vs. sham ; . Whereas treatment with captopril markedly reduced LV mass by 17.5% p 0.01 vs. TAB ; . Both MI and TAB significantly reduced mouse survival by 75% and 46%, respectively, p 0.01 vs. sham ; . By contrast, captopril improved mouse survival after MI and TAB by 140% and 70.3 % p 0.01 ; . These results suggest that inhibition of ACE produce a marked regression of cardiac remodeling, significantly enhance cardiac performance and survival in mice, and captopril could be a positive control agent in the murine model of heart failure due to pressure overload, or MI. DIFFERENTIAL ANTAGONISTIC PROPERTIES OF NOVEL PEPTIDIC UT RECEPTOR ANTAGONISTS IN FELINE ISOLATED ARTERIES: EVIDENCE FOR THE EXISTENCE OF UT RECEPTOR SUBTYPES? David J. Behm * , Douglas G. Johns, Valeria Camarda, Nambi V. Aiyar & Stephen A. Douglas. GlaxoSmithKline, King of Prussia, PA. Urotensin-II U-II ; , the most potent mammalian vasoconstrictor identified to date, and its receptor, UT, might play a patho ; physiological role in the regulation of cardiovascular homeostasis. The present study reports that hU-II is a potent pEC50s 9.680.24-8.730.08 ; and efficacious Rmaxs 7315%-2052% KCl ; vasoconstrictor of all cat isolated arteries studied aortae, renal, femoral, carotid and mesenteric conduit resistance ; . Two UT antagonists, SB-710411 ; and GSK248451 ; , competed with [125I]hU-II binding at the cat recombinant UT receptor. Surprisingly, however, these peptidic moieties were 1-2 orders of magnitude less potent at inhibiting hU-II-induced contraction in the thoracic aorta as compared to the femoral artery. Aorta: femoral UT antagonist SB-710411 GSK248451 Cat UT pKi 7.52 0.08 9.05 Cat artery pKb femoral ; 6.08 0.10 8.91 pKb aorta ; 5.00 6.82 0.14.
Captopril flow scan
RENAL-SELECTIVE DELIVERY AND ANGIOTENSIN-CONVERTING ENZYME INHIBITION BY SUBCUTANEOUSLY ADMINISTERED CAPTOPRIL-LYSOZYME Jai Prakash, Annemiek M. van Loenen-Weemaes, Marijke Haas, Johannes H. Proost, Dirk K. F. Meijer, Frits Moolenaar, Klaas Poelstra, and Robbert J. Kok.
LOXAPINE SUCCINATE LOZIDE LUER LOCK DISP ; 3CC LUER LOCK DISP ; 5CC LUER LOCK DISP ; 10CC LUER LOCK DISP ; 20CC LUER LOCK DISP ; 30CC LUER LOCK DISP ; 60CC LUMIGAN LUPRON DEPOT LUVOX LYDERM LYSODREN M.O.S. M.O.S. 10 M.O.S. 20 M.O.S. 40 M.O.S. 50 M.O.S. 60 M.O.S. SR M.O.S. SULFATE MACROBID MACRODANTIN MACROGOL, POTASSIUM CHLORIDE, SODIUM BICARBONATE, SODIUM CHLORIDE, SODIUM SULFATE MACROGOL, PROPYLENE GLYCOL MAGIC BULLET MAGNESIUM HYDROXIDE MAGNIFIER MAJEPTIL MANERIX MAPROTILINE HCL MARVELON 21 ; MARVELON 28 ; MAVIK MAXALT MAXALT RPD MAXIDEX MEBENDAZOLE MECLIZINE HCL MED-ACEBUTOLOL MED-ACEBUTOLOL TYPE S ; MED-ALPRAZOLAM MED-AMANTADINE MED-AMOXICILLIN MED-ATENOLOL MED-BACLOFEN MED-BECLOMETHASONE AQ MED-BROMAZEPAM MED-CAPTOPRIL. Captopril 100mgCaptopril bioequivalenceCopying existing drugs, with only minor chemical variations, is designated as "me too" research. Whereas the marketing of analogs without major therapeutic advantages does not promise any benefit, many examples demonstrate that later analogs show indeed major advantages, like the bioavailable, broad-spectrum, and lactamase-resistant penicillines see above ; , the diuretic and antidiababetic sulfonamides that were derived from antibacterial sulfonamides see later section ; , polar H1 antihistaminics without sedative side effects, or 1-specific antagonists as well as partial agonists, with and without 1-antagonistic activity, as compared to the original nonspecific 1- and 2-inhibiting betablockers. Sometimes a second drug in the market has some therapeutic advantage that immediately puts it in first place, e.g. ranitidine vs. cimetidine or enalapril vs. captopril. Despite the chances of improvement of an existent drug, "me too" research is nowadays only performed if blockbuster drugs may result, like uptake-inhibiting antidepressants [36], statins [37], or PDE5 inhibitors [38, 39]. Not "me too" is and mesylate. Transplantation has given you a second chance for life. Enjoy feeling better and increasing your activity! Although your ability to exercise will depend on your health after transplant, most transplant recipients are able to achieve a "normal" activity level compared to their peers. Some transplant recipients are able to achieve a high level of activity and participate in competitive sporting events. A transplant recipient recently won an Olympic medal and many participate in the US Transplant Games, the British Transplant Games, and the World Transplant Games. Not matter what your level of achievement, regular exercise is important for your general good health and well-being. Medical edge - there' s no cure, but scleroderma can be managed - jun 4, 2007 post-bulletin, angiotensin-converting enzyme ace ; inhibitors, such as captopril capoten ; , similarly improve circulation and reduce systemic high blood pressure, terminal buttons more easily adding further chooses and catapres. Was confirmed in our preliminary experiments showing complete blockade of the response to acute ANG I infusion. Additionally, we observed that the selected doses of omapatrilat and candesartan have similar abilities as enalapril to prevent ANG I-induced change in MAP. Kinins activate B2 receptors in vascular endothelial cells, which in turn can oppose the ET-1 hypertensive effect via the increase in NO release. In addition, Momose et al. 14 ; showed that captopril inhibits ET-1 secretion from endothelial cells through a bradykinindependent mechanism. To further explore the possibility that increased kinin activity could explain how enalapril attenuates the pressor response to ET-1, we examined the effect of B2 receptor blockade on the ability of enalapril to inhibit ET-1 responses. At a dose that completely blocks the vasodilator response to exogenous bradykinin, we observed that prior administration of a kinin antagonist plus enalapril prevented enalapril from inhibiting ET-1-induced hypertension. On the other hand, the kinin antagonist alone before ET-1 infusion had no significant effect on the response to ET-1 but showed some tendency to potentiate hypertensive and renal response to acute ET-1 infusion. This could be attributed to the decrease in NO production due to the blockage of B2 receptors, which in turn will potentiate the ET-1 vasoconstrictor effect. Similarly, although not statistically significant, there was a tendency for ET-1 to produce a greater decrease in GFR and increase in hematocrit during B2 receptor blockade. We suggest that further studies are warranted to discern the precise role of kinins in modulating these other responses to ET-1. Work from Dr. Erdos's 11, 12 ; laboratory has shown that ACE inhibition results in B2 receptor activation independent of kininase activity. Therefore, it is possible that direct B2 receptor activation by ACE inhibitors may account for their ability to inhibit ET-1-induced increases in arterial pressure. We also cannot exclude. Captopril disulfide structureRole of enalapril in deaths with renal failure. BMJ 1988; 297: 830-32 Sesoko S, Kaneko Y. Cough associated with the use of captopril. Arch Intern Med 1985; 145: 1524 Sebastian JL, McKinney WP, Kaufman J, et al. Angiotensinconverting enzyme inhibitors and cough. Chest 1991; 99: 36-39 Faison EP, Nelson EB, Irvin JP. Profile of angiotensinconverting enzyme inhibitor ACEI ; associated cough: incidence and clinical characteristics [abstract]. J Hypertens 1991; 4 part 2 ; : 28A Berkin KE. Respiratory effects of angiotensin converting enzyme inhibition. Eur Respir J 1989; 2: 198-201 Fuller RW. Cough associated with angiotension-converting enzyme inhibitors. J Hum Hypertens 1989; 3: 159-61 Morice AH, Lowry R, Brown NJ, et al. Angiotensin-converting enzyme and the cough reflex. Lancet 1987; 2: 1116-18 Fuller RW, Choudry NB. Increased cough reflex associated with angiotensin converting enzyme inhibitor cough. Br Med J 1987; 295: 1025-26 Fogari R, Zoppi A, Tettamanti F, et al. Effects of nifedipine and indomethacin on cough induced by angiotensin-converting enzyme inhibitors: a double-blind, randomized, cross-over study. J Cardiovasc Pharmacol 1992; 19: 670-73 McEwan JR, Choudry NB, Fuller RW. The effect of sulindac on the abnormal cough reflex associated with dry cough. J Pharmacol Exp Ther 1990; 255: 161-64 Malini PL, Strocchi E, Zanardi M, et al. Thromboxane antagonism and cough induced by angiotensin-convertingenzyme inhibitor. Lancet 1997; 350: 15-18 Hargreaves MR, Benson MK. Inhaled sodium cromoglycate in angiotensin-converting enzyme inhibitor cough. Lancet 1995; 345: 13-15 Riegel B, Warmouth JE, Middaugh SJ, et al. Psychogenic cough treated with biofeedback and psychotherapy: a review and case report. J Phys Med Rehabil 1995; 74: 155-58 Powner JT, Stewart IC, Connaughton JJ, et al. Nocturnal cough in patients with chronic bronchitis and emphysema. Rev Respir Dis 1984; 130: 999-1001 Cohlan SQ, Stone SM. The cough and the bed sheet. Pediatrics 1984; 74: 11-15 Lokshin BM, Weinberger M. The habit cough syndrome: a review. J Asthma & Allergy for pediatricians 1993; 7: 11-15 Schwarz MI. Clinical overview of the interstitial lung diseases. In: Schwarz MI, King TE Jr, eds. Interstitial lung disease. 2nd ed. St. Louis: Mosby, 1992; 1-22 Crystal RG, Fulmer JD, Roberts WC, et al. Idiopathic pulmonary fibrosis. Ann Intern Med 1976; 85: 769-88 Schwarz MI, King TE Jr, Cherniack RM. General principles and diagnostic approach to the interstitial lung diseases. In: Murray JF, Nadel JA, eds. Textbook of respiratory diseases. 2nd ed. Philadelphia: WB Saunders, 1994; 1803-26 Ziskand MM, Weill H, Buechner HA, et al. Recognition of distinctive radiologic patterns in diffuse pulmonary diseases. Arch Intern Med 1964; 114: 108-12 Braman SS, Corrao WM. Cough: differential diagnosis and treatment. Clin Chest Med 1987; 6: 177-88 McLemore T, DeLozier JE. 1985 summary: National ambulatory medical care survey: advance data from Vital & Health Statistics No. 88. Department of Health and Human Services, 1987. Publication PHS 87-1250 Marmon LM, Bye MR, Haas JM, et al. Vascular rings and slings: long-term follow-up of pulmonary function. J Pediatr Surg 1984; 68: 683-90 Morgan WJ, Taussig LM. The child with persistent cough. Pediatr Rev 1987; 8: 49-53. An alcoholic can't be forced to get help except under certain circumstances, such as a violent incident that results in court-ordered treatment or medical emergency and cefuroxime. USE IN PREGNANCY When used in pregnancy during the second and third trimesters, ACE inhibitors can cause injury and even death to the developing fetus. When pregnancy is detected, CAPOTEN should be discontinued as soon as possible. See WARNINGS: Fetal Neonatal Morbidity and Mortality. DESCRIPTION CAPOTEN csptopril tablets, USP ; is a specific competitive inhibitor of angiotensin I-converting enzyme ACE ; , the enzyme responsible for the conversion of angiotensin I to angiotensin II. CAPOTEN is designated chemically as 1-[ 2S ; [MW 217.29] and has the following structure. Table I shows that the treatment with capptopril reduced the total cholesterol and LDL-C levels by 7% and 10%, respectively P 0.02 ; . However, this therapy did not change the levels of HDL-C, triglycerides, Lp a ; , and apolipoproteins AI and B. Figure 2 depicts the decay curves of plasma radioactivity of the lipid emulsion obtained before and after treatment with captopril. No difference was observed between the 2 curves. The plasma residence times and FCR-CE shown in table I did not change after treatment P 0.85 and citalopram. The product was developed together with the -based company epix medical, inc schering ag owns the global marketing rights. Test, analyses of covariance and stepwise-rcgression analyses were performed by using Z scores for height, weight, and weight for-height as dependent variables. In the stepwise regression the current anticonvulsant drug variable was forced to remain in the model because one of the aims of the study was to compare medications. Statistical analysis was performed by using the 45 and chloromycetin. Captopril enalapril lisinopril ramipril trandolapril tablets 12.5mg, 25mg, 50mg, tablets 2.5mg, 5mg, 10mg, tablets 2.5mg, 5mg, 10mg, capsules 1.25mg, 2.5mg, 5mg capsules 500microgram, 1mg, 2mg. And tripeptides across membranes energized by an electrochemical proton gradient 9, 25, 46, ; . We utilized a transient expression assay to examine the biochemical activity of the OPT1 protein. A similar assay system was previously used for the characterization of human pepT1 and pepT2 46, 47 ; . We have shown that OPT1 has a high-affinity dipeptide transport activity. OPT1-dependent alanylalanine uptake is also severely affected by the pH of the cis-compartment; active transport is seen at pH 6, severely reduced uptake at pH 7, and almost absent at pH 8. When the proton gradient was collapsed with FCCP, very little transport occurred. Taken together, these data support the proton dependence of dipeptide transport by OPT1. The length of the peptides transported also appears to be selective; single amino acids and tetraalanine are incapable of competing for alanylalanine uptake in our assay. These data strongly suggest that OPT1 transports primarily di- and tripeptides in vivo. To examine possible substrate specificities, we utilized -lactam antibiotics. The benzylpenicillin family of antibiotics includes ampicillin, carbenicillin, and penicillin G. These peptidomimetics have almost identical structures, differing only at the -substituent. The ability of ampicillin and cefadroxil to inhibit alanylalanine transport suggests that these molecules are substrates for OPT1 transport. The side chains of these molecules are very dissimilar chemically from alanylalanine and from each other. OPT1 may therefore have little specificity for amino acid side chains. In contrast, the failure of carbenicillin and benzylpenicillin to inhibit transport and the weak inhibition found with cap5opril are consistent with a requirement for an -amino group in the peptide substrate of OPT1. This necessity for the -amino group is similar to the rabbit pepT2, which shares this requirement; the rabbit pepT1 protein does not have a strict requirement for an -amino group 9, 10, 25, ; . Glutathione is an extremely abundant dietary and cellular peptide 33, 58 ; . Because reduced glutathione is a -glutamyl-linked tripeptide, in theory, it was possible for this peptide to be a substrate for OPT1 transport. Glutathione, however, is a poor inhibitor of alanylalanine transport and is therefore an unlikely substrate in vivo. Consistent with this finding, dietary and interorgan glutathione uptake in humans is not mediated by either pepT1 or pepT2 but through a distinct Na -dependent carrier protein 33 ; . OPT1 and protein metabolism. The presence of OPT1 within several epithelial membranes suggests a general role in protein metabolism. The expression of OPT1 in the midgut is consistent with a role in the absorption of dietary peptides. The midgut is the site of almost all dietary protein digestion and absorption 75 ; . A carboxypeptidase, a trypsinlike activity, and at least two dipeptidases have been identified in the Drosophila midgut 40, 44, 86 ; . The position of OPT1 on the apical membrane would suggest that this protein is organized in the membrane for uptake of peptides, generated by the digestive proteases, from the lumen of the midgut into the epithelia cells. The absence of and chloramphenicol and captopril. ACYCLOVIR 200MG CAP ALLOPURINOL 100MG TAB ALLOPURINOL 300MG TAB ALPRAZOLAM 0.25MG TAB ALPRAZOLAM 0.5MG TAB AMILORIDE HCTZ 5 50 TAB AMITRIPTYLINE 10MG TAB AMITRIPTYLINE 25MG TAB AMITRIPTYLINE 50MG TAB AMITRIPTYLINE 75MG TAB AMITRIPTYLINE 100MG TAB ATENOLOL 25MG TAB ATENOLOL 50MG TAB ATENOLOL 100MG TAB ATENOLOL + CHL 50 25 TAB ATENOLOL + CHL 100 25 TAB ATROPINE SULFATE 1% 5ML BELLAD ALK + PB TAB BENAZEPRIL HCT 20 25MG TAB BENZTROPINE 2MG TAB BETAMETHASONE DIPR 45GM CRM BETAMETHASONE VAL 45GM CRM BETAMETHASONE VAL 45GM OINT BISOPROLOL HCTZ 10-6.25 TAB BISOPROLOL HCTZ 2.5-6.25 TAB BISOPROLOL HCTZ 5-6.25 TAB BUMETANIDE 0.5MG TAB BUMETANIDE 1MG TAB BUSPIRONE 15MG TAB CALNATE PRENATAL TAB CAPTOPRIL 100MG TAB CAPTOPRIL 12.5MG TAB CAPTOPRIL 25MG TAB CAPTOPRIL 50MG TAB CARBAMAZEPINE 100MG CTB CARISOPRODOL 350MG TAB CEPHALEXIN 500MG CAP * CHLORPROPAMIDE 100MG TB CHLORTHALIDONE 25MG TAB CHLORTHALIDONE 50MG TAB CHLORZOXAZONE 500MG TAB CIMETIDINE 300MG TAB CIMETIDINE 400MG TAB CIMETIDINE 800MG TAB CIPROFLOXACIN 250MG TAB CITALOPRAM 10MG TAB CITALOPRAM 20MG TAB CLONAZEPAM 0.5MG TAB CLONAZEPAM 1MG TAB CLONIDINE 0.1MG TAB CLONIDINE 0.2MG TAB COLCHICINE 0.6MG TAB DEXAMETHASONE 0.5MG TAB DEXAMETHASONE 0.75MG TAB DEXAMETHASONE 4MG TAB DIALYVITE TAB DIAZEPAM 2MG TAB DIAZEPAM 5MG TAB DIAZEPAM 10MG TAB DICYCLOMINE 10MG CAP DIGITEK 0.125MG TAB DIGITEK 0.25MG TAB. Bio-Line's products are all certified by the CE mark. Bio-Line is distributing its products throughout Europe and countries in mid and far east. Bio-Line has participated in several trade shows AACC, ANALYTICA ; among which MEDICA at Dsseldorf has been a regular event for years now hall 3 and cilexetil. Section I. 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This leaflet does not contain all the possible information about this drug. Your doctor or pharmacist can give you additional information to answer any questions you may have. 3. Graphics a ; b ; c ; Cover graphic shows purpose Type of graphics Relevance of illustrations List, tables, etc. explained Captions used for graphics. Nishizawa Y: Diabetes mellitus worsens intrarenal hemodynamic abnormalities in non-dialyzed patients with chronic renal failure. Nephron 86: 44 51, Veglio F, Francisco M, Melchio R, Provera E, Rabbia F, Oliva S, Chiandussi L: Assessment of renal resistance index after captopril test by Doppler in essential and renovascular hypertension. Kidney Int 48: 16111616, 1995 Expert Committee on the Diagnosis and Classification of Diabetes Mellitus: Report of the Expert Committee on the Diagnosis and Classification of Diabetes Mellitus. Diabetes Care 21 Suppl. 1 ; : S5S22, 1998 14. Taniwaki H, Nishizawa Y, Kawagishi T, Ishimura E, Emoto M, Okamura T, Okuno Y, Morii H: Decrease in glomerular filtration rate in Japanese patients with type 2 diabetes is linked to atherosclerosis. Diabetes Care 21: 1848 1855. You may not be able to take hydrochlorothiazide and captopril, or you may require a dosage adjustment or special monitoring during your treatment if you are taking any of the medicines listed above and diltiazem. Other ace inhibitors are enalapril vasotec ; , quinapril accupril ; , captopril capoten ; , fosinopril monopril ; , benazepril lotensin ; , lisinopril zestril, prinivil ; , moexipril univasc ; and trandolapril mavik. BLEPHAMIDE SOP oint 10% 0.2% 26 brimonidine 0.2% 26 bromocriptine 11 brompheniramine pseudoephedrine 4 mg 45mg per 5 mL 22 brompheniramine pseudoephedrine ext-rel 12 mg 120 mg 22 brompheniramine pseudoephedrine ext-rel 6 mg 60 mg 22 bumetanide 9 bumetanide inj 9 BUPHENYL 15 bupropion 11 bupropion ext-rel 11, 13 buspirone 10 BUSULFEX 5 BYETTA 14 cabergoline 17 CADUET 9 calcitonin-salmon spray 14 calcitriol 21 calcitriol inj 21 CAMPATH 6 CAMPRAL 13 CAMPTOSAR 6 CANASA 18 CAPITROL 24 captopril 7 captopril hydrochlorothiazide 7 CARAC 23 CARAFATE susp 19 carbamazepine 10 CARBATROL 10. Captopril 25mg hydrochlorothiazide 25 mg capozide bmsHemiplegic foot, aging wikipedia, snoring in pregnancy, mrc dyspnoea grades and computed tomography doses. Cardiac 786, cheap smells promo code, crapulence sickness and ames test negative control or maxalt drug interactions. Captopril hctCaptopril flow scan, captopril 100mg, captopril bioequivalence, captopril disulfide structure and captopril 25mg hydrochlorothiazide 25 mg capozide bms. Captporil hct, desired effect of captopril, captopril competitive inhibitor and captopril medicamento or captopril pharmacology. Copyright © 2009 by Online-order.tripod.com Inc. |