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NEW YORK STATE DEPARTMENT OF HEALTH 09 14 2007 LIST OF MEDICAID REIMBURSABLE DRUGS PRICING ERRORS ARE NOT REIMBURSABLE PRICES EFFECTIVE 09 14 2007 MRA COST -0.00870 0.00870 -0.00870 0.10650 -0.00870 0.00870 -0.05500 0.05500 -0.05500 0.05500 COST ALTERNATE -FORMULARY DESCRIPTION MAX STRENGTH SUSP MAALOX MAX STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP MAALOX PLUS X-STRENGTH SUSP PLUS X-STRENGTH SUSP MAALOX QUICK DISSOLVE TAB MAALOX QUICK DISSOLVE TAB MAALOX QUICK DISSOLVE TAB MAALOX QUICK DISSOLVE TAB MAALOX QUICK DISSOLVE TAB MAALOX QUICK DISSOLVE TAB MAALOX QUICK DISSOLVE TAB MAALOX QUICK DISSOLVE TAB MAALOX SUSPENSION SUSPENSION MAALOX SUSPENSION MAALOX SUSPENSION MAALOX SUSPENSION MAALOX SUSPENSION MAG DELAY TABLET SA MAG-G 500 MG TABLET MAG-SR 64 MG TABLET SA MAGGEL 600 MG SOFTGEL MAGNEBIND 200 TABLET 300 TABLET MAGNESIUM OXIDE 400 MG TAB MAGNESIUM OXIDE 420 MG TAB MAGNESIUM OXIDE 500 MG TAB MAGNESIUM 250 MG TABLET MAGNESIUM 30 MG TABLET MAGNESIUM ALUMINUM SUSPENSI MAGNESIUM ALUMINUM SUSPENSI MAGONATE 1 GM 5 SYRUP MAGONATE 27 MG TABLET 400 TABLET MAGOX 400 TABLET MAGOX 400 TABLET MAGTRATE 500 MG TABLET MAPAP CHILD'S 80 MG TAB CHE PA CD -0 0 0 0 0 -0 0 0 0 0 -0 0 0 0 0 -0 0 0 0 0 -0 0 0 0 0.
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The a03 study was designed to evaluate the likeability of lisdexamfetamine dimesylate compared to placebo and two active controls d-amphetamine sulfate 40 mg, a schedule ii stimulant, and diethylpropion hcl 200 mg, a schedule iv stimulant ; in thirty-six volunteers with histories of stimulant abuse. 1.0 mL Serum Collect "peak" specimen at end of IV infusion or 60 minutes after IM injection. Collect "trough" specimen immediately prior to next dose. Record time in hours that have elapsed between last dose and time of specimen collection. Separate serum by centrifugation. Transfer an aliquot to a labelled tube. Store and ship refrigerated. Specimen collected with heparin is unacceptable and cefaclor, for instance, deferoxamine mesylate. On these artificial stimulants. Try eating a more substantial breakfast, followed by a single cup of coffee. This uses the beverage more for enjoyment than for a lift. Using coffee in the right way will allow you to enjoy it. Using it in place of food will not benefit your health. If you can't live without that cup of coffee first thing in the morning and use the stimulants in coffee to get you going, you are likely addicted. TEA HERBAL TEA While many herbal teas are perfectly safe, others contain potent drugs that could prove more hazardous than caffeine. Herbal teas are popular because their range of claims are said to pick you up, calm you down, speed up or slow down the bowels, and even improve your sex life. Suitable pharmaceutically acceptable acids include acetic, benzenesulfonic besylate ; , benzoic, p-bromophenylsulfonic, camphorsulfonic, carbonic, citric, ethanesulfonic, fumaric, gluconic, glutamic, hydrobromic, hydrochloric, hydroiodic, isethionic, lactic, maleic, malic, mandelic, methanesulfonic mesylate ; , mucic, nitric, oxalic, pamoic, pantothenic, phosphoric, succinic, sulfuric, tartaric, p-toluenesulfonic, and the like and cefuroxime. 65 percent of DTC advertisements presented a fair balance of risk and benefit.41 They also found that most ads did not specify how the drug should be used. Another explanation for physicians' resistance is that they do not like losing professional control or are afraid of appearing ignorant or poorly informed. In this view, DTC advertising is just one more rift in the physician's cloak of influence and authority. Ironically, while many physicians oppose DTC advertising, many are themselves regularly influenced by industry sources of information, which can lead to inappropriate prescribing.42 Despite these contradictions, physicians and other prescribing professionals may have legitimate reasons to question the proliferation of DTC advertising. Among the charges leveled by some physicians are that DTC advertising promotes inappropriate prescribing, strains the patient provider relationship, increases the costs of care, and contorts the physician's professional role. In the following sections, we review the evidence for and against these charges. n Effect on prescribing. DTC advertising clearly increases the volume of prescribed drugs. The question is whether these additional prescriptions are appropriate. Using a well-establishe d definition, a prescription is appropriate when "the health benefits of the drug so outweigh the health risks that the drug is worth taking."43 While proponents of DTC advertising claim that such ads avert underuse of effective medicines without greatly increasing inappropriate use, many physicians are wary of this claim, for three reasons. First, as noted, a study from the early 1990s found substantial inaccuracies in advertisements directed at physicians.44 Contrary to the beliefs of many consumers, DTC ads are not subject to mandatory FDA review or approval.45 The market is no more likely to ensure the accuracy of DTC advertisements than of ads in medical journals. Thus, even in the absence of direct empirical evidence, physicians have reason to suspect that DTC ads may contain inaccuracies. Assuming that accurate information is a prerequisite for informed discussion, the specter of inaccuracy is a threat to optimal prescribing. Second, DTC advertising may encourage patients to pressure their physicians to switch them from well-studied treatments to new drugs, for which knowledge about benefits and risks is more limited. In 1991 the American Academy of Pediatrics opposed DTC advertising because it felt that it would encourage demand for treatment not medically indicated and boost inappropriate requests for specific medications.46 The third reason for physicians' skepticism is based on indirect evidence. A generation of geographic variations research has shown that it is very difficult to increase or decrease use of health care. Bioequivalence of Marevan and Coumadin PBAC questions ; - letters 26 2 ; 27-9 dental extractions letter ; 27 1 ; 3 dental patients 25 3 ; 69 - letter 26 4 ; 75-7; 25 5 ; 104-7 interactions with antiplatelet drugs 25 4 ; 81-5 interactions with complementary medicines 25 3 ; 54-6 - letter 26 2 ; 27-9 interactions with fenofibrate 29 6 ; 166 interactions with miconazole 26 2 ; 33-5 risks and benefits 27 4 ; 88-92 - cataract surgery letter ; 27 6 ; 138-41 anticonvulsants - management of behavioural problems in dementia 28 3 ; 67-70 - in neuropathic pain 29 3 ; 72-5 - oxcarbazepine - Trileptal New drug ; 25 1 ; 20-3 - pregabalin - Lyrica New drug ; 28 3 ; 75-9 - withdrawal of antiepileptic drugs in seizure-free adults 27 5 ; 114-7 - withdrawal of drugs from seizure-free children 29 1 ; 18-21 antidepressant drugs - drug-induced hyponatraemia 26 5 ; 114-7 letters 27 2 ; 28-33 - escitalopram oxalate - serotonin reuptake inhibitor - Lexapro New drug ; 26 6 ; 146-51 letter 27 2 ; 28-33 - management of behavioural problems in dementia 28 3 ; 67-70 - management of mild depression in general practice 28 1 ; 8-10 - 'Medicines out of control?' book review ; 27 5 ; 117 - neuropathic pain 29 3 ; 72-5 - psychotropic drugs for children in general practice 28 5 ; 116-8 - reboxetine mesylate - Edronax New drug ; 25 5 ; 120-3 - serotonin syndrome 26 3 ; 62-3; 25 1 ; 19 - suicide risk in children editorials ; 28 5 ; 110-1; 28 5 ; 111-3 letter 29 2 ; 32-5 - treatment of non-depressive disorders 28 4 ; 91-3 - withdrawal of drugs from market editorial ; 26 3 ; 50-1 antiemetics - aprepitant Emend - cancer chemotherapy New drug ; 27 3 ; 76-9 in cancer chemotherapy PBAC question ; 28 5 ; 119 - granisetron - Kytril New drug ; 27 4 ; 101-5 and citalopram. What is factive gemifloxacin mesylate tabletsThe phylogenetic dendrogram based on sequence analysis of the protein kinases catalytic domain Supplementary Figure 2 ; assigns PDGFR and KIT kinases as more divergent from the ABL tyrosine kinase than SRC subfamily. However, Imatinib mesylate exhibits comparable efficiencies and high selectivity at inhibiting PDGFR, KIT and ABL tyrosine kinases, while being largely ineffective in suppressing the tyrosine kinase activity of SRC family Wong and Witte, 2004 ; . The phylogenetic analysis of the protein tyrosine kinases would not have predicted selective inhibition of PDGFR, KIT and ABL tyrosine kinases by Imatinib mesylate without suggesting cross-binding to a broader range of closely related tyrosine kinases Deininger et al., 2005; Wong and Witte, 2004 ; . Hence, functional profile of Imatinib mesylate binding may not be directly linked with the position of protein kinases on the evolutionary dendrogram. Pyrido [2, 3-d]pyrimidine class of inhibitors, including PD-173955 Supplementary Figure 1b ; , were initially developed as broadly active inhibitors of several tyrosine kinases, such as SRC, PDFGR and FGFR receptors Hamby et al., 1997; Boschelli et al., 1998 ; . In subsequent studies, these compounds were also shown to potently inhibit ABL and KIT tyrosine kinases Wong and Witte, 2004; Nagar et al., 2002; Wisniewski et al., 2002 ; . While sensitivity of the tyrosine kinases activity to the PD-173955 inhibitor may not be immediately evident from phylogenetic proximity, all these tyrosine kinases share the evolutionary conserved Thr residue at the gate-keeper site of the binding pocket. Interestingly, although the evolutionary profile of the Thr gatekeeper residue is conserved in all tyrosine kinases from the ABL and SRC families, the respective binding profile of Imatinib mesylate for ABL and SRC kinases can vary dramatically Druker et al., 2004; Lydon and Druker, 2004: Deininger et al., 2005; Wong and Witte, 2004 ; . Although the catalytic domains of protein kinases in the active form are structurally very similar, crystallographic studies have revealed a remarkable plasticity and considerable kinase versatility in adopting vastly different and unique inactive conformations Huse and Kuriyan, 2002 ; , which arguably leads to high selectivity of Imatinib mesylate at inhibiting ABL, KIT and PDGFR tyrosine kinases. A significant body of experimental evidence suggests that protein kinases can occupy a range of natural conformations between inactive and active states and significant differences observed between structures of ABL kinase may be merely a function of the activation state of the ABL kinase Schindler et al., 2000; Wisniewski et al., 2002; Nagar et al., 2002 ; . In the framework of the energy landscape view of molecular recognition, both active and inactive protein conformations may exist in equilibrium which is shifted towards a preferable thermodynamic state upon ligand association. In this work, we conduct in silico profiling of Imatinib m3sylate and PD-173955 kinase inhibitors on kinome scale by using evolutionary analysis and fingerprinting inhibitorprotein interactions with the panel of all publically available protein tyrosine kinases crystal structures. Evolutionary history of the protein tyrosine kinases has led to the conservation patterns, which reflect not only general evolutionary substitution tendencies among amino acids, but are also influenced by the selective pressure on the binding site residues to fulfill structural integrity and functional specificity requirements. We set out to investigate how sequence plasticity of the binding site residues and protein kinase and chloromycetin. Advertised before Acceptance under section 20 1 ; Proviso 1349068 - April 06, 2005. SANVIK LABORATORIES PVT.LTD. A COMPANY INORPORATED UNDER THE COMPANIES ACT 1956. ; 131, RATHI PLAZA, SAMARTH NAGAR, AURANGABAD 431 001. MANUFACTURERS & TRADERS. Address for service in India Agents Address : B. VIJAYALAXMI & CO. 151, VIJAYASHREE BUNGALOW, NR. SAIBABA TEMPLE, JYOTI NAGAR, AURANGABAD-431 005. Proposed to be used. MUMBAI ; ALL KINDS OF MEDICINAL, PHARMACEUTICAL & AYURVEDIC PREPARATIONS & SUBSTANCES, INCLUDED IN CLASS-5, because benztropine mesylate. Whenever queer activists bring up the issue of legal reform, the response of state authorities is inevitably that homosexuality is an offense.23 The HIV AIDS community has also taken up the issue of Section 377, contending it impairs work on HIV AIDS by criminalizing same-sex acts and thereby driving one of the vectors of the disease underground. The National AIDS Control Authority, a governmental agency, has stated in its policy, "Government will review and reform criminal laws and correctional systems to ensure that they are consistent with international human rights obligations and are not misused in the context of HIV AIDS or targeted against vulnerable groups."24 This bold statement, however, has not been followed by any real action to bring about a change in the criminal law. The concerns of these two groups--queer advocates and HIV AIDS activists--intersect in activism around Section 377. Two case studies--the Lucknow case and the constitutional challenge to Section 377--illustrate both the difficulties and opportunities embedded in this activism. Moreover, these case studies demonstrate how an articulation of queer rights can expose the connections of power in the attacks on persons doing HIV AIDS outreach. The Lucknow Case: Health Interventions as Promoting Deviance One of the key problems facing HIV AIDS advocates in the MSM sector is that constant police harassment and surveillance hamper both fieldwork and outreach among the MSM community. This is perhaps best exemplified by the events in Lucknow in 2001. On July 7, 2001, the police, acting on a complaint by a man that he had been sodomized by someone in a park, raided the park that was frequented by hijras, kothis, and others who come within the rubric of queer and arrested 10 people. Among those arrested was an activist from Bharosa an NGO working with the MSM community ; . Thereafter the police raided the offices of Bharosa and Naz Foundation International another NGO working on safer-sex issues ; , seized safer-sex material, and registered a complaint under Section 377 unnatural sexual offenses ; , Section 120B con12 Vol. 7 No. 2 and chloramphenicol. Appendix 3: Texas Wound Chart .87 Appendix 4: Basic Foot Screening Checklist .88 Appendix 5: Measurement of Blood Pressure.89 Appendix 6: Sample Letter for Patients Travelling Abroad .90 Appendix 7: Disposal of Sharps .91 Appendix 8: Referral form for Ethnic Minority Pharmacist Clinic .92 Appendix 9: Lothian Integrated Care Pathway for Diabetic Ketoacidosis.93, for instance, imatinib mesylae india. 4831031 Pfizer Pfizer Pfizer Pfizer Pfizer GlaxoSmithKline GlaxoSmithKline GlaxoSmithKline GlaxoSmithKline SB Pharmco SB Pharmco SB Pharmco SB Pharmco Pfizer Aventis Pharmaceuticals Aventis Pharmaceuticals Aventis Pharmaceuticals Pfizer Pfizer Pfizer Pfizer AstraZeneca AstraZeneca AstraZeneca AstraZeneca AstraZeneca AstraZeneca AstraZeneca AstraZeneca Novartis Novartis Novartis Novartis Novartis Novartis Pfizer Pfizer Pfizer Pfizer Pfizer Pfizer Ziprasidone Hydrochloride eq 20mg base Ziprasidone Hydrochloride eq 40mg base Ziprasidone Hydrochloride eq 60mg base Ziprasidone Hydrochloride eq 80mg base Ziprasidone Mestlate eq 20mg base ml Carvedilol 12.5mg Carvedilol 25mg Carvedilol 3.125mg Carvedilol 6.25mg Carvedilol Phosphate 10mg Carvedilol Phosphate 20mg Carvedilol Phosphate 40mg Carvedilol Phosphate 80mg Estradiol .025mg 24hr Dolasetron Mesylaye eq 100mg base Dolasetron Meylate eq 100mg base 5ml eq 20mg base ml ; Dolasetron Mesyla6e eq 50mg base Glyburide 1.5mg Glyburide 3mg Glyburide 4.5mg Glyburide 6mg Budesonide Budesonide Budesonide Budesonide .16mg 1inh .32mg inh 0.16mg inh base base base base and cilexetil. Croft and Bartsch [41] wrote a comprehensive review on the synthesis of chemically modified CDs. They summarized in their paper the synthesis of acylated, alkylated, deuterated, mesylated, tosylated, and rigidly capped cyclodextrins. Furthermore, the synthesis was described of CDs containing amino, azido, halogen, nitrate, phosphorous, imidazole, pyridine, sulfur, alcohol, aldehyde, keto, oxime, carboxyl, carbonate, carbamate, silicon, boron and tin as functional groups. In this review, we will discuss the synthesis and characterization of those CDs that are in use for pharmaceutical analysis with CE. Several cationic and anionic CDs e.g. sulfobutylether DS 3.5 are SBE[I-VII]-CDs, propylnowadays carboxy m ; ethylated-CDs, sulfated-CDs, mono- 6-[2-hydroxy]. Gerd has been associated with other medical problems, such as: asthma pneumonia bronchitis ear infections with fluid drainage ; if your child has gerd, there is a treatment that can help relieve the painful symptoms and heal the damage sores, also called erosions ; to your child's esophagus and atacand. Barnard PD 1993 ; . National Oral Health Survey Australia 1987-88. Department of Health, Housing, Local Government & Community Services, Canberra: Australian Government Publishing Service. Beck J, Garcia R, Heiss G, Vokonas PS, Offenbacher S 1996 ; . Periodontal disease and cardiovascular disease. J Periodontol 67: 1123-1137. Choi JI, Chung SW, Kang HS, Rhim BY, Kim SJ, Kim SJ 2002 ; . Establishment of Porphyromonas gingivalis heat-shock-proteinspecific T-cell lines from atherosclerosis patients. J Dent Res 81: 344-348. Clauss A 1957 ; . Gerinnungsphysiologische Schnellmethode zur Bestimmung des Fibrinogens [Rapid physiological coagulation method in determination of fibrinogen]. Acta Haematol 17: 237246. D'Aiuto F, Ready D, Tonetti MS 2004 ; . Periodontal disease and Creactive protein-associated cardiovascular risk. J Periodontol Res 39: 236-241. DeStefano F, Anda RF, Kahn HS, Williamson DF, Russell CM 1993 ; . Dental disease and risk of coronary heart disease and mortality. BMJ 306: 688-691. Ebersole JL, Machen RL, Steffen MJ, Willmann DE 1997 ; . Systemic acute-phase reactants, C-reactive protein and haptoglobin, in adult periodontitis. Clin Exp Immunol 107: 347-352. Fey GH, Fuller GM 1987 ; . Regulation of acute phase gene expression by inflammatory mediators. Mol Biol Med 4: 323-338. Fredriksson MI, Figueredo CM, Gustafsson A, Bergstrm KG, sman BE 1999 ; . Effect of periodontitis and smoking on blood leukocytes and acute-phase proteins. J Periodontol 70: 1355-1360. Geerts SO, Nys M, De MP, Charpentier J, Albert A, Legrand V, et al. 2002 ; . Systemic release of endotoxins induced by gentle mastication: association with periodontitis severity. J Periodontol 73: 73-78. Grau AJ, Buggle F, Heindl S, Steichen-Wiehn C, Banerjee T, Maiwald M, et al. 1995 ; . Recent infection as a risk factor for cerebrovascular ischemia. Stroke 26: 373-379. Hamsten A, deFaire U, Walldius G, Dahln G, Szamosi A, Landou C, et al. 1987 ; . Plasminogen activator inhibitor in plasma: a risk factor for recurrent myocardial infarction. Lancet 2: 3-9. Howell TH, Ridker PM, Ajani UA, Hennekens CH, Christen WG 2001 ; . Periodontal disease and the risk of subsequent cardiovascular disease in US male physicians. J Coll Cardiol 37: 445-450. Hujoel PP, Drangsholt M, Spiekerman C, DeRouen TA 2000 ; . Periodontal disease and coronary heart disease risk. J Med Assoc 284: 1406-1410. ALBUTEROL 0.83 MG ML SOLUTION ALBUTEROL 0.83 MG ML SOLUTION IPRATROPIUM BR 0.02% SOLN IPRATROPIUM BR 0.02% SOLN IPRATROPIUM BR 0.02% SOLN ALBUTEROL 5 MG ML SOLUTION VOPAC TABLET CEFUROXIME AXETIL 250 MG TAB CEFUROXIME AXETIL 250 MG TAB CEFUROXIME AXETIL 500 MG TAB CEFUROXIME AXETIL 500 MG TAB CEFACLOR 250 MG CAPSULE CEFACLOR 500 MG CAPSULE CEPHALEXIN 250 MG CAPSULE CEPHALEXIN 250 MG CAPSULE CEPHALEXIN 500 MG CAPSULE CEPHALEXIN 500 MG CAPSULE NUOX GEL FEXOFENADINE HCL 30 MG TABLET FEXOFENADINE HCL 60 MG TABLET FEXOFENADINE HCL 60 MG TABLET FEXOFENADINE HCL 180 MG TABLET FEXOFENADINE HCL 180 MG TABLET OFLOXACIN 0.3% EYE DROPS MIRTAZAPINE 15 MG RPD DISLV TB MIRTAZAPINE 30 MG RPD DISLV TB MIRTAZAPINE 45 MG RPD DISLV TB INNOHEP 20, 000 UNIT ML VIAL INNOHEP 20, 000 UNIT ML VIAL ACYCLOVIR 200 MG CAPSULE ACYCLOVIR 200 MG CAPSULE ACYCLOVIR 400 MG TABLET ACYCLOVIR 400 MG TABLET ACYCLOVIR 800 MG TABLET ACYCLOVIR 800 MG TABLET CEPHALEXIN 250 MG CAPSULE CEPHALEXIN 500 MG CAPSULE CEFACLOR 250 MG CAPSULE CEFACLOR 500 MG CAPSULE CEFACLOR 125 MG 5 ML SUSPEN CEFACLOR 125 MG 5 ML SUSPEN CEFACLOR 187 MG 5 ML SUSPEN CEFACLOR 187 MG 5 ML SUSPEN CEFACLOR 250 MG 5 ML SUSPEN CEFACLOR 250 MG 5 ML SUSPEN CEFACLOR 375 MG 5 ML SUSPEN CEFACLOR 375 MG 5 ML SUSPEN CEPHALEXIN 250 MG CAPSULE CEPHALEXIN 250 MG CAPSULE CEPHALEXIN 250 MG CAPSULE CEPHALEXIN 500 MG CAPSULE DOXAZOSIN MESYLATE 1 MG TAB DOXAZOSIN MESYLATE 1 MG TAB DOXAZOSIN MESYLATE 1 MG TAB DOXAZOSIN MESYLATE 2 MG TAB DOXAZOSIN MESYLATE 2 MG TAB DOXAZOSIN MESYLATE 2 MG TAB DOXAZOSIN MESYLATE 4 MG TAB and candesartan and mesylate. There are variations by the source of infection, with type 1 being more common 60% ; in haemophiliacs than type 3, which is the most common type in IDUs 47% type 1 and 43% type 3 ; . This means that those infected with blood products may respond less well to treatment than those who acquired the virus through drug abuse. Treatment is regarded as successful if blood tests indicating inflammatory liver damage [alanine aminotransferase ALT ; ] return to normal and if the HCV disappears from the blood. A complete response is defined as acceptable ALT levels and no detectable HCV RNA at the end of treatment, and a sustained response constitutes maintenance of these levels for at least 6 months after the treatment has stopped. Early studies used ALT levels and liver histology as outcome measures; later trials added disappearance of the virus, once it could be measured. It is assumed that such measurements indicate response to treatment and if patients respond this will prevent progression of liver disease and development of cirrhosis, portal hypertension, liver failure and possible HCC.14 Those patients with long-term remission and loss of the virus are thought to be unlikely to develop cirrhosis or liver cancer.15 It is recognised that the outcomes used are surrogate markers, but it is still unclear whether a sustained response improves the long-term prognosis for these patients or whether this represents a cure. A recent cohort of 80 patients who had sustained a response to interferon- have been followed for up to 6 years. Response to treatment was maintained and liver histology improved in more than 90% of patients.16. Commonly called a one-sample z-test, this technique compares a proportion obtained from a random sample to a population proportion. It is generally used to determine if there is a difference between what was obtained in a random sample and an established benchmark that either originated from all members of a comparable population or is assumed to represent the population and ciloxan. Award recipients attended the SMRU Postgraduate course: "The Role of the Andrologist in the Era of ART." The course was co-chaired by Peter N. Kolettis, M.D., and Jay I. Sandlow, M.D. The faculty also included Peter N. Schlegel M.D, and Michael P. Steinkampf, M.D., M.P.H. Dr. Andrew R. LaBarbera, the ASRM's Scientific Director, introduced Dr. Benoff and the 2004 award recipients at the Opening Ceremony, and the Scholars attended the Gala Reception which followed. The program also included three morning lectures. These sessions provided small-group format lectures, allowing very personalized and didactic exchanges between the recipients and lecturers. The morning sessions faculty were: Joel L. 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