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See 27, Comment g Tentative Draft No. 2, 2002 ; . It is the latter class of diseases or ones that are treated as such ; that is addressed in this Comment. In this multiple-exposure situation, courts have adopted a rule that each nontrivial exposure to a toxic agent may be found by the trier of fact to be a factual cause of plaintiff's disease. See 27, Comment g, and Reporters' Note thereto Tentative Draft No. 2, 2002 ; . Alternatively, especially when plaintiff has been exposed to the toxic agent of multiple defendants, as is frequently the case with asbestos, plaintiffs would be presented with proving which exposure initiated the disease. If a court adopted the latter requirement, it would then be confronted with the propriety and application of alternative liability, see 28 b ; , and shifting the burden of proof to defendants on the question of factual causation. See Rutherford v. OwensIll., Inc., 941 P.2d 1203, 1215-1220 Cal. 1997 ; . The difference between these two proof requirements results from different assumptions about the biology of disease development. The asbestos rule a "threshold rule" ; rests on an assumption that each dose of asbestos contributes to a threshold dose above which disease is caused in the individual exposed. See Eagle-Picher Indus., Inc. v. Balbos, 604 A.2d 445 Md. Ct. App. 1992 ; expert testifying to belief in an "undefined `threshold' of asbestos exposure" required before disease would occur ; . Alternatively, each dose of a carcinogen may pose an independent and distinct, albeit small, risk of causing cancer in the exposed individual. Indeed, the "one-hit" model of carcinogenesis is consistent with this no-threshold hypothesis. See COMMITTEE ON RISK ASSESSMENT OF HAZARDOUS AIR POLLUTANTS, NATIONAL RESEARCH COUNCIL, SCIENCE AND JUDGMENT IN RISK ASSESSMENT 123-124 1994 COMMITTEE ON THE INSTITUTIONAL MEANS FOR ASSESSMENT OF RISKS TO PUBLIC HEALTH, NATIONAL RESEARCH COUNCIL, RISK ASSESSMENT IN THE FEDERAL GOVERNMENT: MANAGING THE PROCESS 19-20 1983 Joseph V. Rodricks & Susan H. Rieth, Toxicological Risk Assessment in the Courtroom: Are Available Methodologies Suitable for Evaluating Toxic Tort and Product Liability Claims?, 27 REG. TOXICOLOGY & PHARMACOLOGY 21, 23-24 1997. Generic meridia products and generic meridia programs. Wellbutrin bupropion and related ; - rxboard weight gain well vs meridia posted by: d date: friday, 9 february 2001, at 7: 22 i too have heard is one of the few ad that won't cause weight gain.

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Publication history issue online: 17 dec 2006 home list of issues table of contents article abstract annals of the new york academy of sciences volume 761 steroid receptors and antihormones page 355-360, june 1995 to cite this article: fuchs-young, l. Neuronal background of pathological aggression: activational changes in the dorsal raphe Tth Mt, Halsz Jzsef, Haller Jzsef Department of Behavioural Neurobiology, Institute of Experimental Medicine, Budapest matet koki.hu The serotonergic system is well known for its aggression lowering effects. It was recurrently shown, however, that the serotonergic system is activated during fights, and recent data suggested that it is necessary for the expression of aggressive behaviour. We investigated the interaction between serotonergic activation and aggressive behaviour in Wistar rats by assessing the co-localization of the c-Fos signal marker of neuronal activation ; with tryptophan-hydroxylase activity marker of serotonin secretion ; in the raphe. Control animals were compared with animals exposed to visual and olfactory but not physical ; contacts with opponents psychosocial stimulation ; as well as with animals exposed to aggressive encounters. Fights were accompanied by the activation of the raphe; however, the effect was not aggression-specific, as a similar activation was induced by psychosocial contacts. The lack of behavioural specificity in activation suggests that it was related to arousal rather than to aggressiveness. The activation of serotonergic raphe neurons showed a negative correlation with aggressive behaviour, which is in line with the widespread view that serotonin neurotransmission decreases aggression related behavioural phenomena. The activation of serotonergic neurons did not show a correlation with measures of hypoarousal-driven abnormal aggression, which indicates that factors other than the raphe control this behaviour. The latter finding may explain the low efficacy of serotonergic treatments in conduct and antisocial personality disorders, in which violence correlates with hypoarousal and mesterolone. Meridia ordering from actonel our online pharmacy xendos order now and save money.
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