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In addition to ensuring that our medicines policies work today, the development of a new zealand medicines strategy is an opportunity to ensure that with ageing population, changing demographics, rapid advancements in health technologies and treatment options that the system is robust and responsive to these future needs. Downtown Bicycle, a corporation, was not represented by an attorney as required by Alaska law. AAG Dan Branch represented the defendants in the case. ACPE PREVAILS AGAINST TERMINATED EMPLOYEE AAG Mary Ellen Beardsley successfully represented the Alaska Commission on Postsecondary Education in a superior court appeal brought by a terminated former employee. In September 2001, ACPE's executive director terminated Dennis Watson as director of the division of student financial aid claiming he was still a probationary employee who could be terminated without cause. Watson appealed the decision to the commission and a three-member panel was established to consider his appeal. AAG Sarah Felix represented ACPE during this administrative process. After a two day hearing in November 2000, the hearing panel issued a decision upholding the termination. The panel concluded there were sufficient grounds to consider him a probationary employee and therefore, he could be terminated without cause. They also concluded that there was sufficient evidence to support a termination for cause as well. Watson then filed a separate action in the superior court claiming breach of contract and the covenant of good faith and fair dealing. AAG Beardsley took over the case at that point and filed a motion to have the case converted to an administrative appeal. Judge Weeks granted this motion. Oral argument occurred on December 17, 2002, and on December 20th, Judge Weeks issued an opinion upholding the commission's decision. He ruled that the commission has special expertise in determining whether someone is qualified to serve in upper management. He also ruled that the record supported Watson's termination for cause. A motion for attorney's fees is now pending. RCA HAS HEAVY DOCKET AAG Virginia Rusch has been busy assisting the Regulatory Commission of Alaska with an unusual number of court filings, regulations projects, and numerous orders the RCA is issuing to close old dockets and to comply with the first deadline for commission action under the new statute adopted in the legislature's special session last June. AAG Rusch has filed briefs for the RCA in response to two Petitions for Review of commission orders, and three requests for stays. Aurora Energy LLD petitioned for review of the commission order denying an interim increase after Aurora asked for an increase in the rates for electric power it sells to Golden Valley Electric Association under a wholesale power contract. This case involves a first impression interpretation of AS 42.05.431 b ; . The superior court ordered the commission to hold a hearing and consider giving Aurora Energy an interim increase. Golden Valley has now moved for a stay of the superior court's order, which the commission has opposed. In the Trans-Alaska Pipeline intra-state rate case, the commission ordered refunds of excessive rates paid by shippers in the years 1997 through 2000. The carriers have appealed and have filed a motion which argues that under AS 42.06.480 b ; , the commission may not enforce the refund order because they filed their appeal within 10 days. The commission argued that the carriers misinterpret the statute and the judge ruled in the commission's favor. AAG Rusch has also been working with the RCA and legislation and regulations section on several regulations projects. Amendments to 3 AAC 52.900-940, which set terms for pole attachments between utilities, have now been filed with the lieutenant governor's office. Review is ongoing for amendments or new regulations that address the role of the Public Advocacy Section in RCA proceedings, and that adopt new filing requirements and other criteria for interstate interexchange carrier telephone competition, because . Zovirax Ophth Oint 3% Ganciclovir Eye Gel 0.15% Terbinafine HCl Crm 1% Lamisil Crm 1% Amorolfine HCl Nail Laquer Kit 5% 5ml Amorolfine HCl Crm 0.25% Loceryl Nail Laquer Kit 5% 5ml Benzoic Acid Co Oint Clotrimazole Soln 1% Clotrimazole Crm 1% Clotrimazole Pdr 1% Clotrimazole Spy 1% 40ml Canesten Crm 1% Canesten Soln 1% Canesten Dermat Spy 1% 40ml Canesten Pdr 1% Canesten AF Crm 1% Econazole Nit Crm 1% Ecostatin Crm 1% Pevaryl Crm 1% Ketoconazle Crm 2% Nizoral Crm 2% Miconazole Nit Crm 2% Miconazole Nit Dust Pdr 2% Miconazole Nit Pdr Spy 0.16% 100g CFF Daktarin Crm 2% Daktarin Dual Action Pdr 2% Daktarin Dual Action Pdr Spy 0.16% 100g Tioconazole Nail Soln 28.3% Trosyl Nail Soln 28.3% + Applic Nystatin Crm 100, 000u g Nystatin Oint 100, 000u g Nystaform Crm Nystan Crm 100, 000u g Tinaderm M Crm Phytex Paint + Brush. Antifungal Agents: In vitro Susceptibility Testing, Pharmacodynamics, and Prospects for Combination Therapy 573 Andreas H. Groll, M.D. and Hedwig Kolve, Pharm.D. Amphotericin B 591 Astrid M.L. Oude Lashof, M.D. and Bart-Jan Kullberg, M.D. Azoles Clotrimazole, Fluconazole, Itraconazole, Ketoconazole, Miconazole, Voriconazole ; 609 Andreas H. Groll, M.D., Hedwig Kolve, Pharm.D. and Thomas J. Walsh, M.D. Echinocandins Caspofungin ; Alexander Imhof, M.D. and Kieren A. Marr, M.D. Flucytosine 651 Richard H. Drew, Pharm.D. and John R. Perfect, M.D. 637. Canesten AF Pdr 1% Econazole Nit Crm 1% Ecostatin Crm 1% Ketoconazile Crm 2% Nizoral Crm 2% Miconazole Nit Crm 2% Miconazole Nit Dust Pdr 2% Miconazole Nit Pdr Spy 0.16% 100g CFF Daktarin Crm 2% Daktarin Dual Action Pdr Spy 0.16% 100g Tioconazole Nail Soln 28.3% Trosyl Nail Soln 28.3% + Applic Nystatin Crm 100, 000u g Nystatin Oint 100, 000u g Nystan Crm 100, 000u g Phytex Paint + Brush Tinaderm Plus Pdr 1% Mycil Oint Monphytol Paint + Brush Mycota Crm Mycota Pdr Aciclovir Crm 5% Zovirax Crm 5% Zovirax Cold Sore Crm 5% Soothelip Cold Sore Crm 5% Virasorb Cold Sore Crm 5% Herpid Soln 5% Penciclovir Crm 1% Alverine Cit Cap 60mg Alverine Cit Cap 120mg Spasmonal Cap 60mg Sterculia Alverine Gran 62% 0.5% Cisapride Susp 5mg 5ml Prepulsid Tab 10mg Dicycloverine HCl Oral Soln 10mg 5ml Dicycloverine HCl Tab 10mg. Of PBOX-6 on four human breast cancer cell lines. Our results demonstrate that PBOX-6 was a potent inhibitor of both ER-positive MCF-7, IC50 1.9 M; T-47-D, IC50 2 M ; and ER-negative MDA-MB-231, IC50 2.3 M; SK-BR-3, IC50 1 M ; human breast cancer cells. The sensitivities of the four cell lines to PBOX-6 were similar with only a 2-fold difference in the sensitivity observed between the least sensitive MDA-MB-231 ; and most sensitive SK-BR-3 ; cell line. In contrast to these findings, the sensitivity of MDA-MB-231 cells to taxol was 30-fold to that observed for MCF-7 cells 39 ; . This observed difference in taxol sensitivity may be due to factors other than ER status as numerous reports have demonstrated a lack of correlation between cytotoxic chemotherapy and ER status 9, 40 ; . Furthermore, another study again confirmed the lack of correlation between the clinical response to docetaxel and ER status 12 ; . These findings further highlight the potential of PBOX-6 as a chemotherapeutic agent in the management of both hormone-dependent and -independent breast carcinomas. The mechanism of PBOX-6-induced cell death was by apoptosis, as determined by DNA fragmentation pre-G1 peak ; and PARP cleavage. After 48 h treatment, PBOX-6 induced an accumulation of cells in the pre-G1 peak 82.423.10 ; compared with vehicle-treated SK-BR-3 cells 5.940.25 ; . SK-BR-3 cells overexpress the HER-2 oncogene, which is often associated with a poor chemotherapy response 41 ; . As previously discussed, overexpression of the HER-2 oncogene renders breast cancer cells highly resistant to taxol 15 ; . The underlying molecular mechanism is that HER-2 inhibits p34 Cdc2 ; activation, which is required for taxolinduced apoptosis 42 ; . Another study revealed that HER-2 overexpression was also related to poor responsiveness to chemotherapy with cyclophoshamide methotrexate and 5-fluorouracil 42 ; . Given that there was only a 2-fold difference in response to PBOX-6 between the SK-BR-3 and MDA-MB-231 cells, which express low levels of HER-2, PBOX-6 may prove useful as a single agent in the treatment of breast carcinomas overexpressing the HER-2 oncogene. Combination chemotherapy has frequently, but not always produced greater degrees of clinical benefit than single agent therapy. Therefore, it would be interesting to examine the effect of PBOX-6 in combination with other chemotherapeutic agents or hormonal therapy. For example, the combination of paclitaxel with trastuzumab has been shown to be more effective than either drug alone in the treatment of HER-2 overexpressing breast carcinomas 14, 43 ; . Trastuzumab is a humanised monoclonal antibody against the extracellular domain of the HER-2 receptor. Similiarly, increased tumour regressions were also observed in patients with HER-2 overexpressing carcinomas treated with doxorubicin cyclophosphamide supplemented with trastuzumab compared to patients treated with chemotherapy alone 43 ; . In this study, we also showed that PBOX-6 significantly inhibited both the in vitro and in vivo growth of the highly aggressive 4T1 mammary carcinoma cells. No overt toxicity i.e. weight loss ; was apparent in the mice treated with single intratumoural injections of 7.5mg kg PBOX-6 for 7 days. Therefore, it is not unreasonable to assume that higher doses of PBOX-6 may be tolerable and even prove to be more effective at inhibiting tumour growth. However, we found and lamisil. Bourne DW. Clinical and medicoeconomic impact of the cyclosporine-diltiazem interaction in renal transplant recipients. Pharmacotherapy 1994; 14: 471-81. Sketris IS, Methot ME, Nicol D, Belitsky P, Knox MG. Effect of calcium-channel blockers on cyclosporine clearance and use in renal transplant patients. Ann Pharmacother 1994; 28: 1227-31. Keogh A, Spratt P, McCosker C, Macdonald P, Mundy J, Kaan A. Ketoconazolw to reduce the need for cyclosporine after cardiac transplantation. N Engl J Med 1995; 333: 628-33. Jones TE. The use of other drugs to allow a lower dosage of cyclosporin to be used. Therapeutic and pharmacoeconomic considerations. Clin Pharmacokinet 1997; 32: 357-67. Wenke K, Meiser B, Thiery J, et al. Simvastatin reduces graft vessel disease and mortality after heart transplantation: A four-year randomized trial. Circulation 1997; 96: 1398-402.

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By Richard J. Beno CPCU Through the years, SCPIE has handled many cases involving allegations of physician failure to report patients with lapses of consciousness to the local health department. The three cases in this article illustrate the often-dire consequences of failing to follow through on this important step in a patient's medical treatment plan. Although all of these cases take place in California, each state has its own regulations. Physicians should check with their local health department on the reporting requirements for their particular state. In the following random sampling of three cases from our files, a total of four people died and eight were injured. These cases, which totaled more than $3.5 million in defense and indemnity expenses, illustrate instances where death and injury were wholly preventable. Case 1 The patient, a 30-year-old male with a history of juvenile diabetes, first presented to the insured internist in 1979. In 1986, the internist referred the patient to an insured nephrologist because of diabetes-induced renal failure. Later that year, the patient underwent a renal transplant. In 1990, the transplanted kidney was rejected and was subsequently removed. In December 1991, the insured nephrologist hospitalized the patient due to continued complications of diabetes, and requested a consult from a second insured internist. This second internist documented "periods of unconsciousness" in his consultation and sent copies of his report to the nephrologist and the original internist. No Confidential Morbidity Report CMR ; was filed with the local health department. [ Note: In California, pursuant to Health & Safety Code 103900, a CMR must be completed by the diagnosing physician to report patients with "lapses of consciousness." Once the CMR is filed with the local health department, the physician has complied with this statute. The local and lansoprazole, because ketoconazole interaction.

DAVID S. GOLDSTEIN et al. second blood sample was drawn and simultaneous heart rate, blood pressure, stroke volume, and cardiac output was recorded. After the operation, the patient recovered in a semisupine position in the same room that had been used for the baseline observation. Three hours after the operation--sooner if the anesthesia had worn off and the patient complained of moderate to severe pain--the patient underwent a third phlebotomy, filled out the questionnaires, and had vital signs measured manually. The postoperative data collection allowed assessment of biochemical and circulatory interrelationships during pain after the anesthetic effect had worn off. evacuated, heparinized tubes containing no additives. The tubes were spun in a refrigerated centrifuge within 30 min of phlebotomy, and the plasma was transferred into plastic sample tubes, frozen in dry ice, and stored in a tank containing liquid nitrogen. The assay technique used high pressure liquid chromatography with electrochemical detection, the validity and reliability of which have been previously documented in our laboratory 3 ; . Minimum detectable norepinephrine and epinephrine levels were about 15 pg cc. Plasma cortisol, lipids, and lipoproteins were measured using standard laboratory techniques. The total amount of blood drawn per patient, including baseline and both operative sets of samples, was less than 450 ml over a period of at least 2 weeks.

Drug sensitivity of yeast strains was tested by applying serial dilutions start suspension: 1 107 cells mL ; of cell suspensions from overnight cultures, with an applicator, on to solid YPD medium containing different drugs.The plates were incubated for 13 days at 30 C.The drugs, solubilized in adequate solvents sterile water, DMSO, ethanol, or methanol ; were added after the sterilized medium equilibrated at 55 C. A23187 was acquired from Acros Organics; anisomycin, berberine chloride, cerulenin, clotrimazole, daunomycin, dodecyl aldehyde, dodecyl amine, dodecyl arachidate, fluphenazine, ketoconazole, n-lauroyl sarcosine, monensin, nigericin, nystatin, 1, 10-phenantroline, quercetin, rhodamine B, rhodamine 6G, rhodamine 123, SDS, 2, 3, 5-triphenyl tetrazolium chloride tetrazolium red ; , thiazolyl blue tetrazolium bromide tetrazolium blue ; , trifluoperazine, triton X-15, valinomycin, zwittergents 310 and 314, from Sigma; brij 35 from INC Biomedicals Inc.; imazalil, nuarimol from Dr Ehrenstorfer GmbH; cycloheximide and triton X-114 from Nakalai Tesque; curvularol and tautomycin were gifts from H. Osada RIKEN ; . Staurosporine was obtained from Kyowa Hakkou Co. Ltd. For rhodamine B and tautomycin, the pH of the YPD medium was adjusted to 4.5 with hydrochloric acid; for all the other drugs, it was left unchanged. For quantitative assay in liquid culture, cells from overnight cultures were inoculated to the density of 1 105 cells mL in fresh YPD medium containing drugs in two-fold serial dilutions, and grown for 24 h at OD600 was measured for 50-fold dilutions of the cultures and plotted against drug concentration. Minimal inhibitory concentration MIC ; was determined as the lowest drug concentration at which cell growth is nearly completely inhibited Egner et al. 1998 and levofloxacin. Further, we demonstrate that econazole has a MIC of 4 g and a MBC of 4 g while ketoconazole has a MIC of 8 g and a MBC of 16 g ml. Itraconazole, voriconazole, and fluconazole did not inhibit MAC growth to any significant extent. Our studies suggest that inhibition of CYP51 function in MAC may be of therapeutic benefit for patients with this infection.

The cytochrome P450 CYP450 ; enzyme system is responsible for the oxidative-reductive metabolism of medications. These enzymes are primarily concentrated in the liver and small intestines. More than 30 human CYP450 enzymes have been identified; however, only four isoenzymes CYP3A4, CYP2C9, CYP1A2, and CYP2D6 ; are responsible for the majority of drug metabolism. 5 CYP3A4 metabolizes the greatest number of medications and endogenous substances in the body, accounting for most CYP450 enzymes in the liver and small intestines 60 and 70%, respectively ; .6 Many substrates, inhibitors, and inducers of CYP450 isoenzymes have been identified. A substrate is the medication being metabolized by the enzyme system. Warfarin, statins e.g. lovastatin [Mevacor] and simvastatin [Zocor] ; , and theophylline are examples of substrates. An inhibitor is a medication that decreases enzyme activity and leads to increased concentrations of the substrate. Some macrolide antibiotics e.g., erythromycin and clarithromycin [Biaxin] ; , cimetidine Tagamet ; , and azole antifungals e.g., fluconazole [Diflucan], itraconazole [Sporanox], and ketocconazole [Nizoral] ; are known inhibitors of CYP450. An inducer is a medication that increases the number of enzymes and leads to decreased concentrations of the substrate. Notorious enzyme inducers are rifampin Rifadin, Rimactane ; , carbamazepine Tegretol ; , phenytoin Dilantin ; , and phenobarbital. Thus, enzyme inhibitors can predispose to drug toxicity, whereas enzyme inducers have the potential to decrease the effectiveness of a medication. Of the two processes, an interaction that leads to inhibition has a more rapid onset. The optimal management of a drug interaction involves recognition of the interaction; decision regarding whether to prescribe, dispense, or administer the interacting combination; follow-up monitoring; and appropriate patient counseling. Once the interaction is recognized, one must consider the possible outcome before deciding whether to prescribe, dispense, or administer the interacting drugs. If the outcome were possible death, the risk would likely outweigh the benefit. If the outcome were increased drowsiness, the benefit would appear to outweigh the risk and lexapro.

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Discuss your drug regimen with your health care provider if you are prescribed certain antibiotics or antifungals which are not recommended to be taken with simvastatin examples: clarithromycin, erythromycin, itraconazole, ketoconazole.

Buy kehoconazole pills Study details and outcomes data from randomised controlled clinical trials that met the inclusion criteria were collected using a form which was designed for this purpose, then entered into the 'Characteristics of included studies' table in RevMan 4.2.2 by each reviewer sequentially, and were automatically checked for differences. Data was only included when there was agreement. All disagreements were discussed and resolved by consensus. Dunia Alhashimi DAH ; held the master copy. The following details were extracted. 1 ; Study methods: method of allocation, masking of participants and outcomes, exclusion of participants after randomisation and proportion of losses to follow-up. 2 ; Participants: country of origin, sample size, age, sex, inclusion and exclusion criteria. 3 ; Intervention: type of antiemetic; dose, frequency and route. 4 ; Control: placebo or nil. 5 ; Outcomes: any primary and secondary outcomes which had been specified a priori in the 'types of outcomes measures' section of the protocol. 6 ; Adverse effects: any adverse effects related to any clinically diagnosed hypersensitivity or other adverse reactions or side effects to the antiemetics were noted. This information was used to help us assess heterogeneity and the external validity of the trials. Data synthesis Due to significant clinical heterogeneity and the paucity of data in the few included studies, we were unable to do a meta-analysis of the extracted data and therefore only provide a descriptive summary of results of the individual trials. Sensitivity analyses There were insufficient included studies in this systematic review and therefore no attempt was made to conduct a sensitivity analysis and loratadine. Don't take a chance with your health or your life! You can't count on the drug companies to warn you of possible side effects or interactions. Or your doctor or HMO. Or your standard drug reference. But you CAN count on the Graedons, for example, ! Good communication between the medical provider, and close monitoring when starting this drug is crucial and macrodantin.

Ketoconazole cats Does the diet promote a newly discovered fact or secret? Does the diet involve the purchase of a commercial product? Is there a promise of rapid weight loss? Has the diet been independently tested and the results published in a reputable journal? What are the credentials of the author or promoter? Will the diet result in only small amounts of carbohydrates being eaten? Does the diet promote the recommended servings from all food groups? Is there an overemphasis on any one food or food type? Is the energy balance equation considered and, physical activity promoted in conjunction with diet?, for example, keticonazole pills. May seem tedious at first, many providers have found that participating in the chart audit provides a review of the standards of care for HIV AIDS and identifies trends in HIV AIDS care at their facility. Through the audit, the providers often have a better idea of what changes they can make to improve the outcome for people who suffer from this potentially devastating disease. Once the chart audit is complete, the data may be entered into an electronic database, from which you can easily print a summary report. The report shows the percentage of charts having documentation of compliance with each of the indicators. Your AIHA partnership can assist you in obtaining reports and comparison data. In addition, your AIHA partnership can assist you in identifying program strengths and deficiencies. Facilities are encouraged to review the recommendations in a team setting, establish priorities together, and develop an action plan with a timetable for re-evaluation and miconazole. The patient is a 70-year-old white woman who was first admitted to our institution from another hospital secondary to failure to wean from the ventilator. The patient was in her usual state of health until 4 weeks prior to admission to our hospital, when she developed a cough with scant sputum production, chills, nausea, vomiting, and occasional loose stools. Three weeks prior to admission, the patient presented to the referring hospital with a complaint of shortness of breath and she was subsequently admitted. The patient's medical history was significant for hypertension, hyperlipidemia, moderate carotid stenosis, and a remote thalamic infarction 3 years prior to admission, with complete resolution of symptoms. Prior operations included a right eye enucleation for an orbital tumor 20 years prior to admission, and a partial hysterectomy. There was no history of tobacco use or heavy alcohol consumption. Findings from the admitting physical examination were significant for an afebrile patient who was normotensive. Bilateral rhonchi were reported on the physical examination, greatest in the right base. Laboratory data revealed a normal complete blood cell count and platelet count with unremarkable differential. The electrolytes, BUN, creatinine, and phosphorus values were all within normal limits. Hospital admission room air arterial blood gases revealed a pH of 7.42, Pco2 of 44 mm Hg, Po2 of 51 mm Hg, and HCO3 of 28 mEq L. Chest radiograph was performed and showed clear lung fields and normal-sized heart. The patient was admitted to the hospital with the clinical diagnosis of acute pneumonitis and started on a regimen of empiric antibiotic therapy with ceftriaxone. Several days after admission, the patient's condition deteriorated. She developed hypercapnic respiratory failure and lethargy and was transferred to an ICU for monitoring. Two weeks before admission to our hospital, she was intubated for respiratory failure. An arterial blood gas prior to intubation revealed a pH of 7.21, Pco2 of 91 mm Hg, O2 of 50 mm Hg, and an HCO3 of 36 mEq L on 3 min nasal cannula oxygen. A neurologic consultation was sought for continuing respiratory failure and weakness despite unremarkable daily chest radiographs. An electromyogram EMG ; , nerve conduction study, and repetitive stimulation studies were performed and were interpreted as suboptimal but normal. Other studies performed at the referring institution were as follows: normal thyroid-stimulating hormone and vitamin B12; serum protein electrophoresis showed no monoclonal spike; influenza type A viral titer was 1: 64; negative enteroviral serum titers; erythrocyte sedimentation rate was 6 mm h; antinuclear antibody was 1: 80 speckled and normal cerebrospinal fluid protein, glucose, and cell count. The patient was transferred to our hospital for aggressive weaning from the ventilator. She initially spent 1 week in the medical ICU. During her initial stay, the patient failed one extubation attempt and a tracheostomy collar was placed. The patient was transferred to an inpatient ventilation rehabilitation facility. Progressive improvement was noted with regards to both weaning and ambulation at this facility until the fifth week of admission when a deterioration in the patient's status was noted.

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Terconazole is a novel triazole ketal that was synthesized by Janssen Pharmaceutica 129 ; . This compound represents the first triazole antifungal drug marketed for human use. Terconazole is undergoing phase II clinical trials in the United States. Terconazole is available as a 0.8% vaginal cream and pessaries and is indicated for the treatment of vaginal candidiasis Sochynsky and Hardcastle [ed.], Pharma Projects, p. m 596, May 1987 ; . In vitro activity. Terconazole has broad-spectrum antifungal activity in vitro. In one study, 89% of dermatophytes, 100% of Cryptococcus neoformans isolates, and 60% of Candida species were completely or significantly inhibited in vitro by terconazole at a concentration of 10 activity was dependent on the medium used for the assays, however. Terconazole prevented the transformation of C. albicans cells into the pseudohyphal phase in vitro 322 ; . Tolman et al. 304 ; demonstrated that terconazole had a broad range of in vitro antifungal activity, but that the potency varied against isolates of C. albicans and Candida species. These investigators also noted that the anticandidal potency of terconazole was enhanced when the drug was added to yeast cells undergoing mycelial formation. In an assay to test for the suppression of intracellular ATP synthesis, terconazole had no effect. Other tested triazoles also had no effect in this assay 202 ; . Experimental in vivo activity. Terconazole is effective in the treatment of a variety of dermatomycoses and candidiasis in experimental animal models 129, 322 ; . In a guinea pig model, treatment with 0.5% terconazole yielded 10lo cures against infection caused by T. mentagrophytes, and treatment with 1.0% terconazole yielded complete cures against infection caused by M. canis. In an experimental model of rat vaginal candidiasis, terconazole administered twice daily for 3 days as 1.0 and 0.5% creams produced cure rates of 97 and 76%, respectively. In these studies, terconazole also was effective in the treatment of experimental skin candidiasis 322 ; . Terconazole has been shown to have moderate activity following oral administration in experimental vaginal and skin candidiasis and in experimental dermatomycoses 322 ; . Terconazole administered orally at 10 mg kg cured 50% of rats with experimental vaginal candidiasis; oral administration at 40 mg kg cured 67% of animals with experimental M. canis infection. Ketoconszole was superior to terconazole in each of these experiments. Clinical studies. Although clinical studies with terconazole are limited, summary reports of several phase II clinical trials have been published in a special supplement of the journal Gynakolische Rundschau vol. 25, Suppl. 1 ; in 1985. These studies demonstrated the efficacy and tolerability of terconazole in the treatment of vaginal candidiasis. One double-blind study demonstrated that 0.4% terconazole cream was comparable to 1.0% clotrimazole cream in the topical treatment of vaginal candidiasis in a group of 39 pregnant and 40 nonpregnant patients 242 ; . The compound was well tolerated and no serious side effects were reported. The relapse rates of the terconazole- and clotrimazoletreated groups were 10.3 and 17.9%, respectively. A study of similar design also demonstrated the comparable activity of terconazole cream and clotrimazole cream in the treatment of vaginal candidiasis 67 ; . In study of 60 patients with vaginal candidiasis, terconazole 80 mg ; vaginal tablets were compared with clotrimazole 200 mg ; tablets each administered once daily for 3 days or a terconazole 240 mg ; tablet. Nathpong Israngura Na Ayudhya. Prognostic factors of cervical cencer stage IB treated with radical hysterectomy and pelvic lymphadenectomy at Ramathibodi hospital : a multivariate analysis. Bangkok : Mahidol University, 2000. 76 p. T E14832 ; Ponpun Laochariyakul. Funtional study of drug pumps in multidrug resistant cervical cancer cell lines. Bangkok : Mahidol University, 2003. 133 p. T E20499 ; Ratana Sindhuphak. Fourier-transform infrared spectrophotometry [FTIR] : a new approach to cervical cancer detection in Thai women. Bangkok : Institute of Health Research Chulalongkorn University, 2001. 19 p. R E19995 ; Samrit Khahmahpahte. Polymorphisms of PERB 11.2 and the risk to develop cervical cancer. Khon Kaen : Khon Kaen University, 2003. 80 p. T E20671 and monistat and ketoconazole, for example, ketoconazole pregnancy. Morgan Stanley & Co. International Limited, authorised and regulated by Financial Services Authority, disseminates in the UK research that it has prepared, and approves solely for the purposes of section 21 of the Financial Services and Markets Act 2000, research which has been prepared by any of its affiliates. Certain disclosures listed below are also for compliance with applicable regulations in non-US jurisdictions. As of September 30, 2005, Morgan Stanley beneficially owned 1% or more of a class of common equity securities of the following companies covered in this report: Schering AG, GlaxoSmithKline, Merck KGaA, Novartis, Novo Nordisk, Roche, Sanofi-Aventis and Shire Pharma Group. Within the last 12 months, Morgan Stanley managed or co-managed a public offering of securities of Sanofi-Aventis. Within the last 12 months, Morgan Stanley has received compensation for investment banking services from Schering AG, AstraZeneca, Lundbeck, Merck KGaA, Novo Nordisk and Shire Pharma Group. In the next 3 months, Morgan Stanley expects to receive or intends to seek compensation for investment banking services from Schering AG, Altana, AstraZeneca, GlaxoSmithKline, Lundbeck, Merck KGaA, Novartis, Novo Nordisk, Roche, SanofiAventis, Shire Pharma Group and UCB S.A. Within the last 12 months, Morgan Stanley has received compensation for products and services other than investment banking services from Schering AG, AstraZeneca, GlaxoSmithKline, Lundbeck, Novartis and Roche. Within the last 12 months, Morgan Stanley has provided or is providing investment banking services to, or has an investment banking client relationship with, the following companies covered in this report: Schering AG, Altana, AstraZeneca, GlaxoSmithKline, Lundbeck, Merck KGaA, Novartis, Novo Nordisk, Roche, Sanofi-Aventis, Shire Pharma Group and UCB S.A. Within the last 12 months, Morgan Stanley has either provided or is providing non-investment banking, securities-related services to and or in the past has entered into an agreement to provide services or has a client relationship with the following companies covered in this report: Schering AG, Altana, AstraZeneca, GlaxoSmithKline, Lundbeck, Novartis and Roche. The research analysts, strategists, or research associates principally responsible for the preparation of this research report have received compensation based upon various factors, including quality of research, investor client feedback, stock picking, competitive factors, firm revenues and overall investment banking revenues. Morgan Stanley & Co. Incorporated makes a market in the securities of Shire Pharma Group. Morgan Stanley & Co. International Ltd. is a corporate broker to Shire Pharma Group.

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